'Heredity Environment, and The Question How' was originally presented by Anne Anastasi as a presidential address (Division of General Psychology) of the American Psychological Association in 1957.
In this child psychology and nature nurture debate classic, Anastasi argues that the question "How?" offers a much more constructive approach to the heredity-environment problem; as opposed to the question "Which one?" or "How much?" typically posited by psychologists.
Two or three decades ago, the so called heredity-environment question was the center of lively controversy. Today, on the other hand, many psychologists look upon it as a dead issue. It is now generally conceded that both hereditary and environmental factors enter into all behavior. The reacting organism is a product of its genes and its past environment, while present environment provides the immediate stimulus for current behavior. To be sure, it can be argued that, although a given trait may result from the combined influence of hereditary and environmental factors, a specific difference in this trait between individuals or between groups may be traceable to either hereditary or environmental factors alone. The design of most traditional investigations undertaken to identify such factors, however, has been such as to yield inconclusive answers. The same set of data has frequently led to opposite conclusions in the hands of psychologists with different orientations.
Nor have efforts to determine the proportional contribution of hereditary and environmental factors to observed individual differences in given traits met with any greater success. Apart from difficulties in controlling conditions, such investigations have usually been based upon the implicit assumption that hereditary and environmental factors combine in an additive fashion. Both geneticists and psychologists have repeatedly demonstrated, however, that a more tenable hypothesis is that of interaction (15, 22, 28, 40). In other words, the nature and extent of the influence of each type of factor depend upon the contribution of the other.
Thus the proportional contribution of heredity to the variance of a given trait, rather than being a constant, will vary under different environmental conditions. Similarly, under different hereditary conditions, the relative contribution of environment will differ. Studies designed to estimate the proportional contribution of heredity and environment, however, have rarely included measures of such interaction. The only possible conclusion from such research would thus seem to be that both heredity and environment contribute to all behavior traits and that the extent of their respective contributions cannot be specified for any trait. Small wonder that some psychologists regard the heredity environment question as unworthy of further consideration!
But is this really all we can find out about the operation of heredity and environment in the etiology of behavior? Perhaps we have simply been asking the wrong questions. The traditional questions about heredity and environment may be intrinsically unanswerable. Psychologists began by asking which type of factor, hereditary or environmental, is responsible for individual differences in a given trait. Later, they tried to discover how much of the variance was attributable to heredity and how much to environment. It is the primary contention of this paper that a more fruitful approach is to be found in the question "How?" There is still much to be learned about the specific modus operandi of hereditary and environmental factors in the development of behavioral differences. And there are several current lines of research which offer promising techniques for answering the question "How?"
VARIETY OF INTERACTION MECHANISMS
If we examine some of the specific ways in which hereditary factors may influence behavior, we cannot fail but be impressed by their wide diversity. At one extreme, we find such conditions as phenylpyruvic amentia and amaurotic idiocy. In these cases, certain essential physical prerequisites for normal intellectual development are lacking as a result of hereditary metabolic disorders. In our present state of knowledge, there is no environmental factor which can completely counteract this hereditary deficit. The individual will be mentally defective, regardless of the type of environmental conditions under which he is reared.
A somewhat different situation is illustrated by hereditary deafness, which may lead to intellectual retardation through interference with normal social interaction, language development, and schooling. In such a case, however, the hereditary handicap can be offset by appropriate adaptations of training procedures. It has been said, in fact, that the degree of intellectual backwardness of the deaf is an index of the state of development of special instructional facilities. As the latter improve, the intellectual retardation associated with deafness is correspondingly reduced. A third example is provided by inherited susceptibility to certain physical diseases, with consequent protracted ill health. If environmental conditions are such that illness does in fact develop, a number of different behavioral effects may follow. Intellectually, the individual may be handicapped by his inability to attend school regularly. On the other hand, depending upon age of onset, home conditions, parental status, and similar factors, poor health may have the effect of concentrating the individual's energies upon intellectual pursuits. The curtailment of participation in athletics and social functions may serve to strengthen interest in reading and other sedentary activities. Concomitant circumstances would also determine the influence of such illness upon personality development. And it is well known that the latter effects could run the gamut from a deepening of human sympathy to psychiatric breakdown.
Finally, heredity may influence behavior through the mechanism of social stereotypes. A wide variety of inherited physical characteristics have served as the visible cues for identifying such stereotypes. These cues thus lead to behavioral restrictions or opportunities and—at a more subtle level —to social attitudes and expectancies. The individual's own self concept tends gradually to reflect such expectancies. All of these influences eventually leave their mark upon his abilities and inabilities, his emotional reactions, goals, ambitions, and outlook on life.
The geneticist Dobzhansky illustrates this type of mechanism by means of a dramatic hypothetical situation. He points out that, if there were a culture in which the carriers of blood group AB were considered aristocrats and those of blood group O laborers, then the bloodgroup genes would become important hereditary determiners of behavior (12, p. 147). Obviously the association between blood group and behavior would be specific to that culture. But such specificity is an essential property of the causal mechanism under consideration. More realistic examples are not hard to find. The most familiar instances occur in connection with constitutional types, sex, and race. Sex and skin pigmentation obviously depend upon heredity. General body build is strongly influenced by hereditary components, although also susceptible to environmental modification. That all these physical characteristics may exert a pronounced effect upon behavior within a given culture is well known. It is equally apparent, of course, that in different cultures the behavioral correlates of such hereditary physical traits may be quite unlike. A specific physical cue may be completely unrelated to individual differences in psychological traits in one culture, while closely correlated with them in another. Or it may be associated with totally dissimilar behavior characteristics in two different cultures.
It might be objected that some of the illustrations which have been cited do not properly exemplify the operation of hereditary mechanisms in behavior development, since hereditary factors enter only indirectly into the behavior in question. Closer examination, however, shows this distinction to be untenable. First it may be noted that the influence of heredity upon behavior is always indirect. No psychological trait is ever inherited as such. All we can ever say directly from behavioral observations is that a given trait shows evidence of being influenced by certain "inheritable unknowns." This merely defines a problem for genetic research; it does not provide a causal explanation. Unlike the blood groups, which are close to the level of primary gene products, psychological traits are related to genes by highly indirect and devious routes. Even the mental deficiency associated with phenylketonuria is several steps removed from the chemically defective genes that represent its hereditary basis. Moreover, hereditary influences cannot be dichotomized into the more direct and the less direct. Rather do they represent a whole "continuum of indirectness," along which are found all degrees of remoteness of causal links. The examples already cited illustrate a few of the points on this continuum.
It should be noted that as we proceed along the continuum of indirectness, the range of variation of possible outcomes of hereditary factors expands rapidly. At each step in the causal chain, there is fresh opportunity for interaction with other hereditary factors as well as with environmental factors. And since each interaction in turn determines the direction of subsequent interactions, there is an ever-widening network of possible outcomes. If we visualize a simple sequential grid with only two alternatives at each point, it is obvious that there are two possible outcomes in the onestage situation, four outcomes at the second stage, eight at the third, and so on in geometric progression. The actual situation is undoubtedly much more complex, since there will usually be more than two alternatives at any one point.
In the case of the blood groups, the relation to specific genes is so close that no other concomitant hereditary or environmental conditions can alter the outcome. If the organism survives at all, it will have the blood group determined by its genes. Among psychological traits, on the other hand, some variation in outcome is always possible as a result of concurrent circumstances. Even in cases of phenylketonuria, intellectual development will exhibit some relationship with the type of care and training available to the individual. That behavioral outcomes show progressive diversification as we proceed along the continuum of indirectness is brought out by the other examples which were cited. Chronic illness can lead to scholarly renown or to intellectual immaturity; a mesomorphic physique can be a contributing factor in juvenile delinquency or in the attainment of a college presidency! Published data on Sheldon somatotypes provide some support for both of the latter outcomes.
Parenthetically, it may be noted that geneticists have sometimes used the term "norm of reaction" to designate the range of variation of possible outcomes of gene properties (cf. 13, p. 161). Thus heredity sets the "norm" or limits within which environmental differences determine the eventual outcome. In the case of some traits, such as blood groups or eye color, this norm is much narrower than in the case of other traits. Owing to the rather different psychological connotations of both the words "norm" and "reaction," however, it seems less confusing to speak of the "range of variation" in this context.
A large portion of the continuum of hereditary influences which we have described coincides with the domain of somatopsychological relations, as penned by Barker et al. (6). Under this heading, Barker includes "variations in physique that affect the psychological situation of a person by influencing the effectiveness of his body as a tool for actions or by serving as a stimulus to himself or others" (6, p.1). Relatively direct neurological influences on behavior, which have been the traditional concern of physiological psychology, are excluded from this definition, Barker being primarily concerned with what he calls the "social psychology of physique." Of the examples cited in the present paper, deafness, severe illness, and the physical characteristics associated with social stereotypes would meet the specifications of somatopsychological factors.
The somatic factors to which Barker refers, however, are not limited to those of hereditary origin. Bodily conditions attributable to environmental causes operate in the same sorts of somatopsychological relations as those traceable to heredity. In fact, heredity-environment distinctions play a minor part in Barker's approach.
Environmental Factors: Organic
Turning now to an analysis of the role of environmental factors in behavior, we find the same etiological mechanisms which were observed in the case of hereditary factors. First, however, we must differentiate between two classes of environmental influences: (a) those producing organic effects which may in turn influence behavior and (b) those serving as direct stimuli for psychological reactions. The former may be illustrated by food intake or by exposure to bacterial infection; the latter, by tribal initiation ceremonies or by a course in algebra. There are no completely satisfactory names by which to designate these two classes of influences. In an earlier paper by Anastasi and Foley (4), the terms "structural" and "functional" were employed. However, "organic" and "behavioral" have the advantage of greater familiarity in this context and may be less open to misinterpretation. Accordingly, these terms will be used in the present paper.
Like hereditary factors, environmental influences of an organic nature can also be ordered along a continuum of indirectness with regard to their relation to behavior. This continuum closely parallels that of hereditary factors. One end is typified by such conditions as mental deficiency resulting from cerebral birth injury or from prenatal nutritional inadequacies. A more indirect etiological mechanism is illustrated by severe motor disorder—as in certain cases of cerebral palsy—without accompanying injury to higher neurological centers. In such instances, intellectual retardation may occur as an indirect result of the motor handicap, through the curtailment of educational and social activities. Obviously this causal mechanism corresponds closely to that of hereditary deafness cited earlier in the paper.
Finally, we may consider an environmental parallel to the previously discussed social stereotypes which were mediated by hereditary physical cues. Let us suppose that a young woman with mousy brown hair becomes transformed into a dazzling golden blonde through environmental techniques currently available in our culture. It is highly probable that this metamorphosis will alter, not only the reactions of her associates toward her, but also her own self concept and subsequent behavior. The effects could range all the way from a rise in social poise to a drop in clerical accuracy!
Among the examples of environmentally determined organic influences which have been described, all but the first two fit Barker's definition of somatopsychological factors. With the exception of birth injuries and nutritional deficiencies, all fall within the social psychology of physique. Nevertheless, the individual factors exhibit wide diversity in their specific modus operandi - a diversity which has important practical as well as theoretical implications.
Environmental Factors: Behavioral
The second major class of environmental factors—the behavioral as contrasted to the organic—are by definition direct influences. The immediate effect of such environmental factors is always a behavioral change. To be sure, some of the initial behavioral effects may themselves indirectly affect the individual's later behavior. But this relationship can perhaps be best conceptualized in terms of breadth and permanence of effects. Thus it could be said that we are now dealing, not with a continuum of indirectness, as in the case of hereditary and organic-environmental factors, but rather with a continuum of breadth.
Social class membership may serve as an illustration of a relatively broad, pervasive, and enduring environmental factor. Its influence upon behavior development may operate through many channels. Thus social level may determine the range and nature of intellectual stimulation provided by home and community through books, music, art, play activities, and the like. Even more far-reaching may be the effects upon interests and motivation, as illustrated by the desire to perform abstract intellectual tasks, to surpass others in competitive situations, to succeed in school, or to gain social approval. Emotional and social traits may likewise be influenced by the nature of interpersonal relations characterizing homes at different socioeconomic levels. Somewhat more restricted in scope than social class, although still exerting a relatively broad influence, is amount of formal schooling which the individual is able to obtain.
A factor which may be wide or narrow in its effects, depending upon concomitant circumstances, is language handicap. Thus the bilingualism of an adult who moves to a foreign country with inadequate mastery of the new language represents a relatively limited handicap which can be readily overcome in most cases. At most, the difficulty is one of communication. On the other hand, some kinds of bilingualism in childhood may exert a retarding influence upon intellectual development and may under certain conditions affect personality development adversely (2, 5, 10). A common pattern in the homes of immigrants is that the child speaks one language at home and another in school, so that his knowledge of each language is limited to certain types of situations. Inadequate facility with the language of the school interferes with the acquisition of basic concepts, intellectual skills, and information. The frustration engendered by scholastic difficulties may in turn lead to discouragement and general dislike of school. Such reactions can be found, for example, among a number of Puerto Rican children in New York City schools (3). In the case of certain groups, moreover, the child's foreign language background may be perceived by himself and his associates as a symbol of minority group status and may thereby augment any emotional maladjustment arising from such status (34).
A highly restricted environmental influence is to be found in the opportunity to acquire specific items of information occurring in a particular intelligence test. The fact that such opportunities may vary with culture, social class, or individual experiential background is at the basis of the test user's concern with the problem of coaching and with "culture-free" or "culture-fair" tests (cf. 1, 2). If the advantage or disadvantage which such experiential differences confer upon certain individuals is strictly confined to performance on the given test, it will obviously reduce the validity of the test and should be eliminated.
In this connection, however, it is essential to know the breadth of the environmental influence in question. A fallacy inherent in many attempts to develop culture-fair tests is that the breadth of cultural differentials is not taken into account. Failure to consider breadth of effect likewise characterizes certain discussions of coaching. If, in coaching a student for a college admission test, we can improve his knowledge of verbal concepts and his reading comprehension, he will be better equipped to succeed in college courses. His performance level will thus be raised, not only on the test, but also on the criterion which the test is intended to predict. To try to devise a test which is not susceptible to such coaching would merely reduce the effectiveness of the test. Similarly, efforts to rule out cultural differentials from test items so as to make them equally "fair" to subjects in different social classes or in different cultures may merely limit the usefulness of the test, since the same cultural differentials may operate within the broader area of behavior which the test is designed to sample.
The examples considered so far should suffice to highlight the wide variety of ways in which hereditary and environmental factors may interact in the course of behavior development. There is clearly a need for identifying explicitly the etiological mechanism whereby any given hereditary or environmental condition ultimately leads to a behavioral characteristic—in other words, the "how" of heredity and environment. Accordingly, we may now take a quick look at some promising methodological approaches to the question "how."
Within the past decade, an increasing number of studies have been designed to trace the connection between specific factors in the hereditary backgrounds or in the reactional biographies of individuals and their observed behavioral characteristics. There has been a definite shift away from the predominantly descriptive and correlational approach of the earlier decades toward more deliberate attempts to verify explanatory hypotheses. Similarly, the cataloging of group differences in psychological traits has been giving way gradually to research on changes in group characteristics following altered conditions.
Among recent methodological developments, we have chosen seven as being particularly relevant to the analysis of etiological mechanisms. The first represents an extension of selective breeding investigations to permit the identification of specific hereditary conditions underlying the observed behavioral differences. When early selective breeding investigations such as those of Tryon (36) on rats indicated that "maze learning ability" was inherited, we were still a long way from knowing what was actually being transmitted by the genes. It was obviously not "maze learning ability" as such. Twenty - or even ten - years ago, some psychologists would have suggested that it was probably general intelligence. And a few might even have drawn a parallel with the inheritance of human intelligence.
But today investigators have been asking: Just what makes one group of rats learn mazes more quickly than the other? Is it differences in motivation, emotionality, speed of running, general activity level? If so, are these behavioral characteristics in turn dependent upon group differences in glandular development, body weight, brain size, biochemical factors, or some other organic conditions? A number of recent and ongoing investigations indicate that attempts are being made to trace, at least part of the way, the steps whereby certain chemical properties of the genes may ultimately lead to specified behavior characteristics.
An example of such a study is provided by Searle's (31) follow-up of Tryon's research. Working with the strains of maze-bright and maze-dull rats developed by Tryon, Searle demonstrated that the two strains differed in a number of emotional and motivational factors, rather than in ability. Thus the strain differences were traced one step further, although many links still remain to be found between maze learning and genes. A promising methodological development within the same general area is to be found in the recent research of Hirsch and Tryon (18). Utilizing a specially devised technique for measuring individual differences in behavior among lower organisms, these investigators launched a series of studies on selective breeding for behavioral characteristics in the fruit fly, Drosophila. Such research can capitalize on the mass of available genetic knowledge regarding the morphology of Drosophila, as well as on other advantages of using such an organism in genetic studies.
Further evidence of current interest in the specific hereditary factors which influence behavior is to be found in an extensive research program in progress at the Jackson Memorial Laboratory, under the direction of Scott and Fuller (30). In general, the project is concerned with the behavioral characteristics of various breeds and cross-breeds of dogs. Analyses of some of the data gathered to date again suggest that "differences in performance are produced by differences in emotional, motivational, and peripheral processes, and that genetically caused differences in central processes may be either slight or non-existent" (29, p. 225). In other parts of the same project, breed differences in physiological characteristics, which may in turn be related to behavioral differences, have been established.
A second line of attack is the exploration of possible relationships between behavioral characteristics and physiological variables which may in turn be traceable to hereditary factors. Research on EEC, autonomic balance, metabolic processes, and biochemical factors illustrates this approach. A lucid demonstration of the process of tracing a psychological condition to genetic factors is provided by the identification and subsequent investigation of phenylpyruvic amentia. In this case, the causal chain from defective gene, through metabolic disorder and consequent cerebral malfunctioning, to feeblemindedness and other overt symptoms can be described step by step (cf. 32; 33, pp. 389-391). Also relevant are the recent researches on neurological and biochemical correlates of schizophrenia (9). Owing to inadequate methodological controls, however, most of the findings of the latter studies must be regarded as tentative (19).
Prenatal environmental factors provide a third avenue of fruitful investigation. Especially noteworthy is the recent work of Pasamanick and his associates (27), which demonstrated a tie-up between socioeconomic level, complications of pregnancy and parturition, and psychological disorders of the offspring. In a series of studies on large samples of whites and Negroes in Baltimore, these investigators showed that various prenatal and paranatal disorders are significantly related to the occurrence of mental defect and psychiatric disorders in the child. An important source of such irregularities in the process of childbearing and birth is to be found in deficiencies of maternal diet and in other conditions associated with low socioeconomic status. An analysis of the data did in fact reveal a much higher frequency of all such medical complications in lower than in higher socioeconomic levels, and a higher frequency among Negroes than among whites.
Direct evidence of the influence of prenatal nutritional factors upon subsequent intellectual development is to be found in a recent, well controlled experiment by Harrell et al. (16). The subjects were pregnant women in low income groups, whose normal diets were generally quite deficient. A dietary supplement was administered to some of these women during pregnancy and lactation, while an equated control group received placebos. When tested at the ages of three and four years, the offspring of the experimental group obtained a significantly higher mean IQ than did the offspring of the controls.
Mention should also be made of animal experiments on the effects of such factors as prenatal radiation and neonatal asphyxia upon cerebral anomalies as well as upon subsequent behavior development. These experimental studies merge imperceptibly into the fourth approach to be considered, namely, the investigation of the influence of early experience upon the eventual behavioral characteristics of animals. Research in this area has been accumulating at a rapid rate. In 19 54, Beach and Jaynes (8) surveyed this literature for the Psychological Bulletin, listing over 130 references. Several new studies have appeared since that date (e.g., 14, 21, 24, 25, 35). The variety of factors covered ranges from the type and quantity of available food to the extent of contact with human culture. A large number of experiments have been concerned with various forms of sensory deprivation and with diminished opportunities for motor exercise. Effects have been observed in many kinds of animals and in almost all aspects of behavior, including perceptual responses, motor activity, learning, emotionality, and social reactions
In their review, Beach and Jaynes pointed out that research in this area has been stimulated by at least four distinct theoretical interests. Some studies were motivated by the traditional concern with the relative contribution of maturation and learning to behavior development. Others were designed in an effort to test certain psychoanalytic theories regarding infantile experiences, as illustrated by studies which limited the feeding responses of young animals. A third relevant influence is to be found in the work of the European biologist Lorenz (23) on early social stimulation of birds, and in particular on the special type of learning for which the term "imprinting" has been coined. A relatively large number of recent studies have centered around Hebb's (17) theory regarding the importance of early perceptual experiences upon subsequent performance in learning situations. All this research represents a rapidly growing and promising attack on the modus operandi of specific environmental factors.
The human counterpart of these animal studies may be found in the comparative investigation of child-rearing practices in different cultures and subcultures. This represents the fifth approach in our list. An outstanding example of such a study is that by Whiting and Child (38), published in 1953. Utilizing data on 75 primitive societies from the Cross-Cultural Files of the Yale Institute of Human Relations, these investigators set out to test a number of hypotheses regarding the relationships between child-rearing practices and personality development. This analysis was followed up by field observations in five cultures, the results of which have not yet been reported (cf. 37).
Within our own culture, similar surveys have been concerned with the diverse psychological environments provided by different social classes (11). Of particular interest are the study by Williams and Scott (39) on the association between socioeconomic level, permissiveness, and motor development among Negro children, and the exploratory research by Milner (26) on the relationship between reading readiness in first-grade children and patterns of parent-child interaction. Milner found that upon school entrance the lowerclass child seems to lack chiefly two advantages enjoyed by the middle-class child. The first is described as "a warm positive family atmosphere or adult-relationship pattern which is more and more being recognized as a motivational prerequisite of any kind of adult-controlled learning." The lower-class children in Milner's study perceived adults as predominantly hostile. The second advantage is an extensive opportunity to interact verbally with adults in the family. The latter point is illustrated by parental attitudes toward mealtime conversation, lower-class parents tending to inhibit and discourage such conversation, while middle-class parents encourage it.
Most traditional studies on child-rearing practices have been designed in terms of a psychoanalytic orientation. There is need for more data pertaining to other types of hypotheses. Findings such as those of Milner on opportunities for verbalization and the resulting effects upon reading readiness represent a step in this direction. Another possible source of future data is the application of the intensive observational techniques of psychological ecology developed by Barker and Wright (7) to widely diverse socioeconomic groups.
A sixth major approach involves research on the previously cited somatopsychological relationships (6). To date, little direct information is available on the precise operation of this class of factors in psychological development. The multiplicity of ways in which physical traits—whether hereditary or environmental in origin—may influence behavior thus offers a relatively unexplored field for future study.
The seventh and final approach to be considered represents an adaptation of traditional twin studies. From the standpoint of the question "How?" there is need for closer coordination between the usual data on twin resemblance and observations of the family interactions of twins. Available data already suggest, for example, that closeness of contact and extent of environmental similarity are greater in the case of monozygotic than in the case of dizygotic twins (cf. 2). Information on the social reactions of twins toward each other and the specialization of roles is likewise of interest (2). Especially useful would be longitudinal studies of twins, beginning in early infancy and following the subjects through school age. The operation of differential environmental pressures, the development of specialized roles, and other environmental influences could thus be more clearly identified and correlated with intellectual and personality changes in the growing twins.
Parenthetically, I should like to add a remark about the traditional applications of the twin method, in which persons in different degrees of hereditary and environmental relationships to each other are simply compared for behavioral similarity. In these studies, attention has been focused principally upon the amount of resemblance of monozygotic as contrasted to dizygotic twins. Yet such a comparison is particularly difficult to interpret because of the many subtle differences in the environmental situations of the two types of twins. A more fruitful comparison would seem to be that between dizygotic twins and siblings, for whom the hereditary similarity is known to be the same. In Kallmann's monumental research on psychiatric disorders among twins (20), for example, one of the most convincing bits of evidence for the operation of hereditary factors in schizophrenia is the fact that the degrees of concordance for dizygotic twins and for siblings were practically identical. In contrast, it will be recalled that in intelligence test scores dizygotic twins resemble each other much more closely than do siblings—a finding which reveals the influence of environmental factors in intellectual development.
The heredity-environment problem is still very much alive. Its viability is assured by the gradual replacement of the questions, "Which one?" and "How much?" by the more basic and appropriate question, "How?" Hereditary influences— as well as environmental factors of an organic nature—vary along a "continuum of indirectness." The more indirect their connection with behavior, the wider will be the range of variation of possible outcomes. One extreme of the continuum of indirectness may be illustrated by brain damage leading to mental deficiency; the other extreme, by physical characteristics associated with social stereotypes. Examples of factors falling at intermediate points include deafness, physical diseases, and motor disorders. Those environmental factors which act directly upon behavior can be ordered along a continuum of breadth or permanence of effect, as exemplified by social class membership, amount of formal schooling, language handicap, and familiarity with specific test items.
Several current lines of research offer promising techniques for exploring the modus operandi of hereditary and environmental factors. Outstanding among them are investigations of: (a) hereditary conditions which underlie behavioral differences between selectively bred groups of animals; (b) relations between physiological variables and individual differences in behavior, especially in the case of pathological deviations; (c) role of prenatal physiological factors in behavior development; (d) influence of early experience upon eventual behavioral characteristics; (e) cultural differences in child-rearing practices in relation to intellectual and emotional development; (f) mechanisms of somatopsychological relationships; and (g) psychological development of twins from infancy to maturity, together with observations of their social environment. Such approaches are extremely varied with regard to subjects employed, nature of psychological functions studied, and specific experimental procedures followed. But it is just such heterogeneity of methodology that is demanded by the wide diversity of ways in which hereditary and environmental factors interact in behavior development.
1. ANASTASI, ANNE. Psychological testing. New York: Macmillan, 1954.
2. ANASTASI, ANNE. Differential psychology. (3rd ed.) New York: Macmillan, 1958.
3. ANASTASI, ANNE, & CORDOVA, F. A. Some effects of bilingualism upon the intelligence test performance of Puerto Rican children in New York City. J. educ. Psychol, 1953, 44, 1-19.
4. ANASTASI, ANNE, & FOLEY, J. P., JR. A proposed reorientation in the heredityenvironment controversy. Psychol. Rev., 1948, 55, 239-249.
5. ARSENIAN, S. Bilingualism in the postwar world. Psychol. Bull, 1945, 42, 65-86.
6. BARKER, R. G., WRIGHT, BEATRICE A., MYERSON, L., & GONICK, MOIXIE R. Adjustment to physical handicap and illness: A survey of the social psychology of physique and disability. Soc. Sci. Res. Coun. Bull., 1953, No. 55 (Rev.)
7. BARKER, R. G., & WRIGHT, H. F. Midwest and its children: The psychological ecology of an American town. Evanston, 111.: Row, Peterson, 1955.
8. BEACH, F. A., & JAYNES, J. Effects of early experience upon the behavior of animals. Psychol. Bull., 1954, 51, 239-263.
9. BRACKBIIX, G. A. Studies of brain dysfunction in schizophrenia. Psychol. Bull., 1956, 53, 210-226.
10. DARCY, NATALIE T. A review of the literature on the effects of bilingualism upon the measurement of intelligence. G. genet. Psychol., 1953, 82, 21-57.
11. DAVIS, A., & HAVIGHURST, R. J. Social class and color differences in child rearing. Amer. social. Rev., 1946, 11, 698-710.
12. DOBZHANSKY, T. The genetic nature of differences among men. In S. Persons (Ed.), Evolutionary thought in America. New Haven: Yale Univer. Press, 1950. Pp. 86-155.
13. DOBZHANSKY, T. Heredity, environment, and evolution. Science, 1950, 111, 161-166.
14. FORGUS, R. H. The effect of early perceptual learning on the behavioral organization of adult rats. J. comp. physiol. Psychol., 1954, 47, 331-336.
15. HALDANE, J. B. S. Heredity and politics. New York: Norton, 1938.
16. HARRELL, RUTH F., WOODYARD, ELLA, & GATES, A. I. The effect of mothers' diets on the intelligence of the offspring. New York: Bur. Publ., Teach. Coll., Columbia Univer., 1955.
17. HEBB, D. O. The organization of behavior. New York: Wiley, 1949.
18. HIRSCH, J., & TRYON, R. C. Mass screening and reliable individual measurement in the experimental behavior genetics of lower organisms. Psychol. Bull., 1956, 53, 402-410.
19. HORWITT, M. K. Fact and artifact in the biology of schizophrenia. Science, 1956, 124, 429-430.
20. KALLMANN, F. J. Heredity in health and mental disorder; Principles of psychiatric genetics in the light of comparative twin studies. New York: Norton, 1953.
21. KING, J. A., & GURNEY, NANCY L. Effect of early social experience on adult aggressive behavior in C57BL10 mice. J. comp. physiol. Psychol., 1954, 47, 326-330.
22. LOEVINGER, JANE. On the proportional contributions of differences in nature and in nurture to differences in intelligence. Psychol. Bull., 1943, 40, 725-756.
23. LORENZ, K. Der Kumpan in der Unwell des Vogels. Der Artgenosse als aus-16'sendes Moment sozialer Verhaltungsweisen. J. Orn., Lpz., 1935, 83, 137-213; 289-413.
24. LUCHINS, A. S., & FORGUS, R. H. The effect of differential postweaning environment on the rigidity of an animal's behavior. J. genet. Psychol., 1955, 86, 51-58.
25. MELZACK, R. The genesis of emotional behavior: An experimental study of the dog. J. comp. physiol. Psychol., 1954, 47, 166-168.
26. MILNER, ESTHER A. A study of the relationships between reading readiness in grade one school children and patterns of parent-child interaction. Child Develpm., 1951, 22, 95-112.
27. PASAMANICK, B., KNOBLOCH, HILDA, & LILIENFELD, A. M. Socioeconomic status and some precursors of neuropsychiatric disorder. Amer. J. Orthopsychiat., 1956, 26, 594-601.
28. SCHWESINGER, GLADYS C. Heredity and environment. New York: Macmillan, 1933.
29. SCOTT, J. P., & CHARLES, MARGARET S. Some problems of heredity and social behavior. J. gen. Psychol., 1953, 48, 209-230.
30. SCOTT, J. P., & FULLER, J. L. Research on genetics and social behavior at the Roscoe B. Jackson Memorial Laboratory, 1946-1951 - A progress report. J. Hered., 1951, 42, 191-197.
31. SEARLE, L. V. The organization of hereditary maze-brightness and maze-dullness. Genet. Psychol. Monogr., 1949, 39, 279-325.
32. SNYDER, L. H. The genetic approach to human individuality. Sci. Man., N. Y., 1949, 68, 165-171.
33. SNYDER, L. H., & DAVID, P. R. The principles of heredity. (5th ed.) Boston: Heath, 1957.
34. SPOERL, DOROTHY T. Bilinguality and emotional adjustment. J. abnorm. soc. Psychol, 1943, 38, 37-57.
35. THOMPSON, W. R., & MELZACK, R. Early environment. Sci. Amer., 1956, 194 (1), 38-42.
36. THYON, R. C. Genetic differences in mazelearning ability in rats. Yearb. mat. Soc. Stud. Educ., 1940, 39, Part I, 111-119.
37. WHITING, J. W. M., et al. Field guide for a study of socialization in five societies. Cambridge, Mass.: Harvard Univer., 1954 (mimeo.).
38. WHITING, J. W. M., & CHILD, I. L. Child training and personality: A cross-cultural study. New Haven: Yale Univer. Press, 1953.
39. WILLIAMS, JUDITH R., & SCOTT, R. B. Growth and development of Negro infants: IV. Motor development and its relationship to child rearing practices in two groups of Negro infants. Child Develpm., 1953, 24, 103-121.
40. WOODWORTH, R. S. Heredity and environment: A critical survey of recently published material on twins and foster children. Soc. Sci. Res. Coun. Bull., 1941, No. 47.
Nov 26, 23 10:14 AM
Nov 24, 23 05:29 AM
Nov 22, 23 10:10 AM